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INTRODUCTION
Chlamydia trachomatis is the most common bacterial agent of sexually transmitted genital infections. A significant proportion of patients are asymptomatic, thereby providing an ongoing reservoir for infection. In infants born to mothers through an infected birth canal, conjunctivitis and pneumonia can occur. These syndromes are caused by the C. trachomatis serovars B and D through K. The L serovars cause lymphogranuloma venereum, a genital ulcer syndrome. (See "Lymphogranuloma venereum".)
The microbiology, pathophysiology, epidemiology and clinical genital C. trachomatis infections in women, including during pregnancy, will be reviewed here. Pelvic inflammatory disease, lymphogranuloma venereum, genital chlamydia infections in men, and endemic trachoma, an ocular infection caused by serovars A to C that is spread by direct contact and seen commonly in the developing world, are discussed separately. (See "Clinical features and diagnosis of pelvic inflammatory disease" and "Lymphogranuloma venereum" and "Genital Chlamydia trachomatis infections in men" and "Epidemiology, diagnosis, and management of trachoma".)
MICROBIOLOGY
C. trachomatis is a small gram-negative bacterium with unique biologic properties that distinguish it from all other living organisms. It is an obligate intracellular parasite that has a distinct life-cycle that consists of two major phases:
Chlamydia cannot be cultured on artificial media; traditionally tissue culture has been required to established a diagnosis. Rapid and inexpensive screening tests are now available. (See "Screening for Chlamydia trachomatis".)
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