Cerebral venous thrombosis (CVT) is an uncommon but serious disorder. Clinical manifestations can include headache, papilledema, visual loss, focal or generalized seizures, focal neurologic deficits, confusion, altered consciousness, and coma.
Many cases have been linked to inherited and acquired thrombophilias, pregnancy, puerperium, infection, and malignancy. Infarctions due to CVT are often hemorrhagic and associated with vasogenic edema. (See "Etiology, clinical features, and diagnosis of cerebral venous thrombosis".)
Treatment, which is started as soon as the diagnosis is confirmed, consists of reversing the underlying cause when known, control of seizures and intracranial hypertension, and antithrombotic therapy. Anticoagulation is the mainstay of acute and subacute treatment for CVT.
This topic will review the prognosis and treatment of CVT. Other aspects of this disorder are discussed separately. (See "Etiology, clinical features, and diagnosis of cerebral venous thrombosis".)
Cerebral venous thrombosis (CVT) can result in death or permanent disability, but usually has a favorable prognosis.